Cell Signaling Irak-M Antibody

List Price $132.31 Your Price $132.31
Cell Signaling Irak-M Antibody - CSIG (Additional S&H or Hazmat Fees May Apply)
NETA PART: CSIG-4369T
MFG.PART: 4369T
UNSPSC: 12352203
Manufacturer: Cell Signaling
Size20 µl
ReactivityH Mk
SensitivityEndogenous
Molecular Weight (kDa)68
Source/IsotypeRabbit
Application/Dilution{Western Blotting: 1:1000}
StorageSupplied in 10 mM sodium HEPES (pH 7.5), 150 mM NaCl, 100 µg/ml BSA and 50% glycerol. Store at –20°C. Do not aliquot the antibody.
Specificity/SensitivityIRAK-M antibody detects endogenous levels of total IRAK-M protein. Cross reactivity was not detected with other family members.
Species ReactivityHuman, Monkey
Source/PurificationPolyclonal antibodies are produced by immunizing animals with a synthetic peptide corresponding to residues near the carboxy-terminus of human IRAK-M. Antibodies were purified by protein A and peptide affinity chromagraphy.
BackgroundInterleukin-1 (IL-1) receptor-associated kinase (IRAK) is a serine/threonine-specific kinase that can be coprecipitated in an IL-1-inducible manner with the IL-1 receptor (1). The mammalian family of IRAK molecules contains four members (IRAK1, IRAK2, IRAK3/IRAK-M, and IRAK4). The binding of IL-1 to IL-1 receptor type I (IL-1RI) initiates the formation of a complex that includes IL-1RI, AcP, MyD88, and IRAKs (2). IRAK undergoes autophosphorylation shortly after IL-1 stimulation. The subsequent events involve IRAK dissociation from the IL-1RI complex, its ubiquitination, and its association with two membrane-bound proteins: TAB2 and TRAF6. The resulting IRAK-TRAF6-TAB2 complex is then released into the cytoplasm where it activates protein kinase cascades, including TAK1, IKKs, and the stress-activated kinases (3).Unlike IRAK1 and IRAK4, IRAK2 and IRAK-M do not have significant kinase activity although they can still activate NF-κB when overexpressed (4). Expression of IRAK-M is more restricted compared to other family members with highest levels of expression occurring in monocytes/macrophages (4). Studies from IRAK-M knockout mice suggest that IRAK-M may play a role as a negative regulator of Toll-like receptor signaling and innate immune responses by preventing the dissociation of IRAK1 and IRAK4 from MyD88 and the subsequent formation of its complex with TRAF6 (5).
More Information
SKU CSIG-4369T
Featured No
Supplier Part Number 4369T
UM EA
UNSPSC 12352203
Manufacturer Name Cell Signaling
MSDS URL Click here
Temperature -20C
CountryOfOrigin United States
ProductLine CSIG
Qty 1
MinOrderQty 1
Weight 7.000000
Lead Time 5
Hazardous N
ACT Ecolabel No
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